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Tricuspid stenosis
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## π **TRICUSPID STENOSIS**
### π« **Overview**
* **Tricuspid stenosis (TS)** often **coexists with mitral stenosis (MS)**.
* In the illustrated diagram, **both MS and TS** are shown.
* β **Blood flow** entering the **left ventricle (LV)**
* β **Effort intolerance** (fatigue, dyspnea)
### βοΈ **Pathophysiology**
* **β Left atrial (LA) pressure** β Pulmonary edema β Dyspnea on exertion
* **Long-standing pulmonary edema** leads to:
* Pulmonary artery hypertension (**PAH**)
* Right ventricular hypertrophy (**RVH**)
* β Diastolic pressure in the **RV**
β‘οΈ TS acts like a **barrier**, preventing backward pressure transmission due to its narrow orifice β sparing the RV.
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### π’ **Clinical Features**
* **Dyspnea on exertion**
* **Orthopnea** (due to pulmonary congestion)
* **Pulmonary artery hypertension (PAH)**
* **Right-sided congestive heart failure** after **percutaneous mitral balloon valvotomy**:
* Hepatomegaly
* Persistent abdominal fullness
* Right upper quadrant pain
* Ascites
* Pedal edema
> β οΈ *If untreated, may progress to **cardiac cirrhosis.***
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### π©Ί **On Examination**
* **Large βaβ waves** β RA working against high pressure
* **Blunted βyβ descent** β Reduced blood flow through the narrowed tricuspid valve
* **Presystolic pulsations** of the liver
* **Mid-diastolic murmur**, louder on inspiration
* Best heard at **left lower sternal border**
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## π¬ **Investigations (Work-up)**
### π **ECG**
* **Large P wave (P-pulmonale)** β Peaked P wave
* Limb leads: > 2.5 mm
* Chest leads: > 1.5 mm
* **Right axis deviation**
### π©» **ECHO**
* **Thickened tricuspid valve**
* *Severe if orifice area < 1 cmΒ²*
* *Mitral stenosis severe if < 1.5 cmΒ²*
* **Prolonged transit time:** > 190 msec
### π« **Chest X-Ray**
* Right atrial enlargement
* Prominent superior vena cava
* Enlarged shadow of azygos vein
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## π **Treatment**
* π§ **Salt-restricted diet**
* π§ **Diuretics**
* π§ **Valve repair** β *Main treatment*
* βοΈ **Prosthetic valve replacement**
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